For women with hirsutism, acne, or ARQ 197 third-generation progestins desogestrel dyslipidemia.3 and norgestimate, have been con Us, a progestin obtained Hte activity t retained but reduced androgenic activity t. With less androgenic activity of t, the estrogen component st To express themselves more strongly metabolically.3 This can be effective for acne, but increased estrogen hte expression of it To h Run a higher risk for thromboembolic events, particularly in which COC with EE.3, 8 The fourth generation of the progestin, drospirenone and were con DNG We specifically bind to the progesterone receptor without interaction with other stero Several of receptors.8 innovations over the years have introduced in COC formulations and packaging. The terms k Can a function Dependence on the type and amount of hormones, the reasons for these quantities over the cycle, and the number of active pills in the package. Formulations were monophasic active pills contain the same amount of estrogen and progestin. Multiphasic formulations have different amounts of estrogen and progestin, which vary. Biphasic formulations have two different combinations of estrogen and progestin, and three-phase pills have 3 different combinations. Recently a four-phasic formulation developed with a step down and the estrogen-progestin-step, most CoC packages sequence.9 21 active pills and 7 placebo pills contain these packages are known as 21/7 patterns . W Occurs during the week placebo withdrawal bleeding. Early in the development of the COC, it was good because at the time of pregnancy tests were not available quickly, and he assured women they were not pregnant. Than doses of estrogen and progestin decreased in COCs, decreasing serum levels low enough that the H Utung of the endometrium within 2 to 3 days after the last active pill.3 begins Reduce the number of placebo tablets in low-dose formulations necessary to ensure the recruitment of follicles 10.11 Therefore, several formulations in shorter intervals such bare hormones are now prevented in 24/4 or 26/2 diagrams. As the COC has evolved, it became clear that individual women to individual needs receivers Ngnisverhütung have. more than 38 million women in the U.S. Twenty-eight percent of them practice contraception.12 w choose their prime re aCOCas isfect method.12 The pill is to suppress ovulation and gonadotropins to. The estrogen component prevents a Erh Increase in hormone production folliclestimulating to prevent dominant follicle to develop, w While inhibiting the luteinizing hormone progestin on the prevention ovulation.13, thwart COC ovulation by negative feedback to the hypothalamus -pituitary gonadotropin-releasing hormone reduced by Pulsatilit t and reduced pituitary response to GnRH stimulation.3 estrogen helps also lead to unwanted proliferation of the endometrium and outflow minimize and reinforcing RKT the effect of component progestin. The progestin prevents the mitotic effects of estrogen, A steady decidualized building Caused rmutterschleimhaut is not recommended Accessible for implantation.13, 14 In addition, progestins cause a thickening of the cervical mucus to prevent the transport of sperm of. The combination of these mechanisms may provide contraceptive efficacy. Perfect use of COC was sh.
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